A nurse is educating a client with peptic ulcer disease (PUD)regarding Helicobacter pylori and its role in PUD.Which statements by the nurse are appropriate to include in the teaching session? Select all that apply.
A) "The bacteria produces enzymes that improves the efficacy of mucous gel in protecting the gastric mucosa."
B) "Helicobacter pylori infection is found in about 25% of individuals with PUD."
C) "Your inflammatory response to H. pylori contributes to gastric cell damage."
D) "H. pylori infection increases production of gastric acids."
E) "H. pylori infection is spread by droplets in the air."

Question

Quizplus · Accepted Answer

A) Helicobacter pylori infection, found in about 70% of individuals who have PUD, is unique in colonizing the stomach. It is spread individual to individual (oral-oral or fecal-oral) and contributes to ulcer formation in several ways. The bacteria produce enzymes that reduce the efficacy of mucous gel in protecting the gastric mucosa. In addition, the host's inflammatory response to H. pylori contributes to gastric epithelial cell damage without producing immunity to the infection. H. pylori infection increases the production of gastric acids.
B) Helicobacter pylori infection, found in about 70% of individuals who have PUD, is unique in colonizing the stomach. It is spread individual to individual (oral-oral or fecal-oral) and contributes to ulcer formation in several ways. The bacteria produce enzymes that reduce the efficacy of mucous gel in protecting the gastric mucosa. In addition, the host's inflammatory response to H. pylori contributes to gastric epithelial cell damage without producing immunity to the infection. H. pylori infection increases the production of gastric acids.
C) Helicobacter pylori infection, found in about 70% of individuals who have PUD, is unique in colonizing the stomach. It is spread individual to individual (oral-oral or fecal-oral) and contributes to ulcer formation in several ways. The bacteria produce enzymes that reduce the efficacy of mucous gel in protecting the gastric mucosa. In addition, the host's inflammatory response to H. pylori contributes to gastric epithelial cell damage without producing immunity to the infection. H. pylori infection increases the production of gastric acids.
D) Helicobacter pylori infection, found in about 70% of individuals who have PUD, is unique in colonizing the stomach. It is spread individual to individual (oral-oral or fecal-oral) and contributes to ulcer formation in several ways. The bacteria produce enzymes that reduce the efficacy of mucous gel in protecting the gastric mucosa. In addition, the host's inflammatory response to H. pylori contributes to gastric epithelial cell damage without producing immunity to the infection. H. pylori infection increases the production of gastric acids.
E) Helicobacter pylori infection, found in about 70% of individuals who have PUD, is unique in colonizing the stomach. It is spread individual to individual (oral-oral or fecal-oral) and contributes to ulcer formation in several ways. The bacteria produce enzymes that reduce the efficacy of mucous gel in protecting the gastric mucosa. In addition, the host's inflammatory response to H. pylori contributes to gastric epithelial cell damage without producing immunity to the infection. H. pylori infection increases the production of gastric acids.

A Nurse Is Educating a Client with Peptic Ulcer Disease