If protein kinase A is activated in a liver cell in response to glucagon binding to the $\beta$2-adrenergic receptor, which of the following will result?
A) Glycogen synthesis will be turned on.
B) Glycogen degradation will be turned on.
C) Glucose synthesis will be turned off.
D) GLUT1 expression will be upregulated.

Question

Quizplus · Accepted Answer

Activation of protein kinase A in a liver cell in response to glucagon binding to the F1F1F1S1F1F1F102-adrenergic receptor will result in glycogen degradation being turned on. This is because protein kinase A activates enzymes involved in glycogen breakdown, promoting the release of glucose into the bloodstream. Glycogen synthesis would be turned off as protein kinase A inhibits the enzymes involved in glycogen synthesis. Glucose synthesis would be turned on in response to glucagon, but this is not directly related to protein kinase A activation. GLUT1 expression is not directly regulated by protein kinase A in liver cells.

If Protein Kinase a Is Activated in a Liver Cell β\betaβ

If Protein Kinase a Is Activated in a Liver Cell $\beta$